Maternal Acetonuria Not Found Harmful

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T cells is found to be more prominent in hyper-emesis than in women with an uncomplicated pregnancy12. Thus hyperactivation of the maternal immune system may be responsible for the onset of hyperemesis, probably while maternal immune tolerance to the semi-allograft is being established. Various hormonal and immunological factors

Hyperemesis Gravidarum: A Review - e-mjm.org

with maternal blood11,12. These are destroyed by the hyperactivated maternal immune system. The functional activation of natural killer and cytotoxic T-cells is found to be more prominent in hyperemesis than in women with an uncomplicated pregnancy13. The clinical severity

child's fourth year (Herschkowitz, 1976). The time of

deprivation of maternal food, and maternal diabetes, acetonuria, albuminuria with or without hypertension, and infections, among other factors, are known to affect human development. These en-dogenous factors may delay prenatal development and the ontogenesis of the nervous system of the fetal brain (Pan American Health Or-ganization, 1969).