Polymorphism Of The Cardiac Manifestations In Dermatomyositis

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Prognostic Factors of Juvenile Dermatomyositis Introduction

However, the presence of systemic manifestations and the short- and long-term side effects of the treatment may adversely affect morbidity and mortality. While somatic development, functional ability, and educational achievement are usually normal, some patients still have a chronic disease. Aims of the study

CONCISE REPORT Possible role for tumour necrosis factor

formation of new ones.3 An association of dermatomyositis with a 2308A TNFa polymorphism has been reported.4 Serum levels of soluble TNF receptors 1 and 2 were raised in patients with active dermatomyositis or polymyositis when compared with those in controls5 or in patients with inactive disease.6 Increased TNFamRNA expression in muscle biopsy

Lyme disease the trigger of dermatomyositis (a case study)

Dermatomyositis, a myopathic inflammatory disease, characterized by specific skin lesions, with exceptionally, cardiac involvement. DR7 persons, with polymorphism of the tumoral necrotic

Sertraline-Induced Rhabdomyolysis, Trismus, and Cardiac

Aug 04, 2018 Dermatomyositis with concomitant sertraline-induced rhabdomyolysis, renal failure, trismus, and cardiac arrest.‍ DISCUSSION Rhabdomyolysis is a rare complication of SSRI therapy in adults and has been reported, independently of SS, 6 times in the setting of sertraline use.‍9 13 However, to our knowledge, it has not yet been


dermatomyositis complicated with calcinosis. J Rheumatol 33 (8): 1691-1693, 2006 (IF: 3.010) 25. Isomoto S, Kawakami A, Arakaki T, Yamashita S, Yano K, Ono K: Effects of antiarrhythmic drugs on apoptotic pathways in H9c2 cardiac cells. J Pharmacol Sci 101 (4): 318-324, 2006 (IF: 1.792) 26.

Mosaic Tetrasomy 9p: A Mendelian Condition Associated With

juvenile DM (dermatomyositis) or polymyositis with another autoimmune disease, such as SLE, scleroderma, juvenile idiopathic arthritis, inflammatory bowel disease, type 1 diabetes mellitus, or celiac disease.14 An increasing number of Mendelian conditions predisposing to the development of pediatric SLE have been identified.1,2 To date,

Retrospective study of dermatomyositis. Discussion of 40

Dermatomyositis (DM) is included in idiopathic infl am-matory myopathies, together with ),1-6 polymyositis (PMs although exclusively cutaneous clinical forms without muscu-lar involvement are recognized for a variable period of time, and even defi nitely amyopathic (ADM) forms.7-15 It is characterized by the involvement of upper limb proxi-

Mixed connective tissue disease - DUO

DM Dermatomyositis dsDNA double-stranded DNA EMG Electromyography ERS European Respiratory Society ESC European Society of Cardiology FEV1 forced expired volume in one second FVC forced vital capacity GERD Gastroesophageal reflux disease GC Glucocorticoids GN Glomerulonephritis Hgb Hemoglobin HLA human leukocyte antigen

Cacoub P. (2014) Extrahepatic manifestations of chronic

tis/dermatomyositis; PAN, polyarteritis nodosa. 2. type Immune-related extrahepatic manifestations 2.1. Cryoglobulinaemia vasculitis Mixed cryoglobulinaemia (MC) vasculitis is a small vessel vas-culitis involving mainly the skin, the joints, the peripheral nerve system and the kidneys. HCV infection represents the cause of MC in roughly 80% of

Cutaneous Connective Tissue Diseases: Epidemiology, Diagnosis

fasciitis, along with more common entities like Sjogren s syndrome, dermatomyositis, scleroderma, and lupus erythema-tosus. The latter three will be the focus of this review, as they constitute the most significant and common CTD with cuta-neous manifestations.

The Rheumatism Society of the District of Columbia

gastrointestinal manifestations (nausea, left upper quadrant pain, and constipation or diarrhea), polyarthralgia, and sacroiliitis. The course of her disease was complicated by an episode of isolated ACTH insufficiency that resolved after treatment with hydrocortisone and fludrocortisone for 3 years. At the

Rheumatology E-learning University of Szeged Department of

system, cardiac) During active disease: corticosteroid (12-64 mg methylprednisolon) + azathioprin, methotrexate, cyclosporine, mycophenolate mofetil In remission: taper off corticosteroids, maintain other immunosuppressants for 3-5 years (+ antimalarial, Vitamin D3 lifelong)

Polymyositis and dermatomyositis

0501 for juvenile dermatomyositis,14 or tumour necrosis factor 308A polymorphism for photosensitivity in dermatomyositis.15 Emerging information on the genetic background of various ethnic groups may allow identification of immune-response genes that predispose certain populations to polymyositis or dermatomyositis.16,17