Role Of Atrial Natriuretic Peptide In Hemoconcentration During Exercise

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The Regulation and Measurement of Plasma Volume in Heart Failure

Atrial natriuretic peptide, origi-nating in the atria, and brain natriuretic peptide, released from the ventricular myocardium in response to wall stress (21), are structurally related peptides with important natri-uretic and diuretic actions (20,22). An increase in the glomerular filtration rate arises from increased perfusion

Review Article Hypoxia regulates the natriuretic peptide system

name Atrial Natriuretic Factor (ANF). Many labo-ratories subsequently competed to be the first to reveal the structure of ANF, as a result of which several groups published its structure in mammals within a short period of time in 1983-84 [2-8]. The factor was a peptide (later re-ferred to as Atrial Natriuretic Peptide, ANP) and

Hypovolemia and reduced hemoglobin mass in patients with

natriuretic peptide (NT-proBNP > 125 pg mL 1), rele-vant structural/functional cardiac alterations, and no his-tory of iron deficiency (ID) anemia. Healthy age- and sex-matched individuals were recruited from the commu-nity and excluded if they had any chronic medical illness, were taking daily prescription medications, had current

Skeletal Muscle Compensation for Cardiac Muscle Insufficiency

he was referred for invasive exercise testing as part of a transplant evaluation workup. Laboratories revealed hemoglobin 13.3 g/dL, creatinine 1.5 mg/dL, sodium 132 mmol/L, troponin 0.06 ng/mL, and NT-proBNP (N-terminal pro-B-type natriuretic peptide) 14 080 pg/mL. Echocardiography showed ejection fraction 24%, left ven-

NASA-CR-205327 Progress Report and Statement of Work

14. Watenpaugh DE, CW Yancy, JC Buckey, LD Lane, AR Hargens, and CG Blomqvist. Role of atrial natriuretic peptide in systemic responses to acute isotonic volume expansion. Journal of Applied Physiology 73:1218-1226, 1992. 15. Crenshaw AG, JR Sty/, and AR Hargens. Intramuscular pressures during exercise: An evaluation of

Exercise as a stressor to the human neuroendocrine system

the hormone (8). During an exercise bout MCR can vary either through changes in target tissue uptake or hormonal degradation. Additionally, for some hormo-nes the binding affinities of carrier protein change during exercise thereby making more or less of the hormonal free form available in the circulation (10).