Effect Of Nitrous Oxide Anesthesia Upon Cerebral Energy Metabolism
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Failure of flunarizine to improve cerebral blood flow or
FLUNARIZINE IN COMPLETE CEREBRAL SCHEM AJNewberg et al 667 Methods Twelve unmedicated fasting adult mongrel dogs, weighing 10-15 kg were studied. Six dogs were used to study the effect of flunarizine on CBF an2d CMR0 after 10 min of complete cerebral ischemia. Anesthesia for the surgical preparatio 1 %n wa halothans e in 60-70% nitrous oxide
Brain protection during neurosurgery
record, nitrous oxide possesses undesirable characteristics. Nitrous oxide increases CBF, which could cause problems in patients with increased intracra-nial pressure [34,35]. When used alone, nitrous oxide can increase CBF by 37% and cerebral metabolism . When used in combination with inhalational
Reduction of Cerebrospinal Fluid Pressure by Hypocapnia
kg) were studied. Anesthesia was induced with halothane (> 1.3% inspired) in nitrous oxide (N20, 60-70%) and ox ygen. The trachea was intubated, and ventilation was controlled to maintain initial blood gases (Radiometer BMS3 Mk2 electrodes) at Pao2 > 120 mm Hg and Paco2 35-40 mm Hg. The right femoral artery was cannulated
Anaesthetics safe in neonates Proposal for update of Section
These differences have important implications for drug effect, loading dose, interval of dosing and drug metabolism. A premature infant has only ~ 18% of weight as muscle, a term infant ~ 30%, a 6-month-old ~ 40%, and most children >one year ~ 50%. If giving a medication that has its primary effect at the myoneural junction, one could
Effects of Phenobarbital in Cerebral Ischemia
SUMMARY Recovery of cerebral energy metabolism, following 15 or 30 min of pronounced, incomplete ischemia, was studied after 90 min of recirculation in rats that were either anesthetized wit 70%h N2O or 150 mg kg'1 of phenobarbital. In all animals arterial blood pressurea an, Pod Pco, were close to normal during recirculation. In nitrous
Anesthesia Implications for Patients with MELAS
MELAS AND ANESTHESIA 6 Upon arrival to the operating room, the child was induced via a facemask with 70% nitrous oxide, 30% oxygen, and sevoflurane. After induction a combination of air/ oxygen with sevoflurane was titrated to allow for a sufficient anesthetic depth. A 24-gage IV was inserted
Caloric restriction impedes age-related decline of
tracheotomized, and ventilated (30% oxygen; B68% nitrous oxide). After surgery anesthesia was maintained with a-chloralose (initial 80mg/kg, plus 40mg/kg/hour) and D-tubocurarine-Cl (0.3mg/kg) was administered for immobilization. The left femoral artery and vein were catheterized for continuous monitoring of arterial blood pressure and blood
EVALUATION WITH INVASIVE AND NONINVASIVE TECHNIQUES
Arterial (A) and venous (V) concentrations of nitrous oxide during inhalation of 15% nitrous oxide by human subjects. Samples were taken from a jugular bulb catheter (V) and from a peripheral artery catheter (A). (Redrawn from Kety SS, Schmidt CF: The determination of cerebral blood flow in man by use of nitrous oxide in low concentrations.
Oxidative Metabolismin Fetal RatBrain During Maternal
lated with fetal oxidative metabolism and the energy state ofthe brain. Forcomparison, cerebral metabolismin fetuses ofpentobarbital anesthetized damsalso wasinvestigated. Methods Termpregnantrats wereanesthetizedwithether, tracheostomized, andparalyzed with tubocurare (3 mg/kg). Animals were artificially ventilated with 70% nitrous oxide and
9. Anlagen - Der Habilitationsschrift zugrunde liegende
Anesthesia was induced by an intramuscular injection of ketamine hydrochloride (20 mg/kg body wt.) and midazolam (1 mg/kg body wt.) and inhalation of 70% nitrous oxide in 30% oxygen.