Coronary Arteriographic Findings In Patients With Previous Acute Myocardial Infarction

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R Kammler et al, J Clin Case Rep 21, :1 Journal of Clinical

the rarest congenital anomalies (0.02-0.07% of all patients undergoing coronary angiography), especially if originating from the left coronary sinus [1-4]. In these patients, the single coronary artery arises from the aortic trunk through a single ostium and supplies the entire heart. Yamanaka and Hobbs modified the previous model of Lipton et al.

The electrocardiogram in the acute coronary syndromes

patients with nontransmural myocardial infarction. All ofthe patients had >75% narrowing in at least one coronary artery. 28% had triple and 32% had double vessel disease. These findings were similar to those found in patients with stable angina. leading theauthors to emphasize that syndromes of angina. unstable angina and myocardial infarction

Access To And Availability Of Coronary Artery Bypass Grafting

patients with coronary artery disease coronary artery disease cad is narrowing or blockage of one or for acute coronary syndrome 234 53 dual antiplatelet therapy

Exercise-Induced S.T Segment Elevation

a normal coronary arteriogram remains well and pain-free. COMMENTS Elevation of the S-T segment is recognized as a frequent electrocardiographic abnormality in the acute stage of transmural myocardial infarction but is less commonly ob- served as a manifestation of transient myocardial ischemia.

Morphological basis of radiological shadows chest radiographs

pulmonary infarction, subsegmental atelectasis, and septal fibrosis singly and in combination. Potentially transient shadows were seen in association with atelectasis and pulmonary oedema. Band shadows are linear intrapulmonary shadows

Double Left Anterior Descending Coronary Artery Originating

ouble left anterior descending (LAD) coronary artery, with one originating from the left main stem and another originating from the right coronary artery or right aortic sinus, is an extremely rare congenital coronary anomaly.1 The incidence of double LAD ranges from 0.01 to 0.03%. 2 In the present case report, double LAD coronary

Sudden Unexpected Death as a Result of Anomalous Origin of

cardial infarction, has been previously implicated as a cause of myocardial ischemiaiinfarction. Cheitlin et al [l] described a 30-year-otd man with a documented inferior wall myocardial infarction in whom both the right as well as the left main coronary arteries originated from the left sinus of Valsalva.

Left descending electrocardiographic, angiographic correlates

myocardial infarction was made on the basis of a patients (Table4). IngroupA66percent ofthose clear history ofmyocardial infarction accompanied witha definite myocardialinfarction, 80percent of bytypical acute electrocardiographic changes anda those witha possible myocardial infarction, and82 rise in serum enzymes. per cent ofthose

Not all ST-segment elevation myocardial infarctions become Q

fied as having an ST-segment elevation myocardial infarction, and in the absence of a contraindication, they receive reperfusion therapy, either thrombolysis or angioplasty. Without reperfusion, most such patients will develop Q waves and have sizable infarcts. All other patients with acute coronary syndromes are said to have unstable angina.

Coronary Artery Involvement in Takayasu Arteritis in 45

finding of coronary involvement as baseline. The clinical features, laboratory data, coronary angio-graphic findings, treatment, and followup outcomes were summarized retrospectively. Results.A total of 45 (7.7%, 45/587) patients with coronary involvement were identified, including 40 with typical angina, and 15 with myocardial infarction.

Coronary Arteriographic Lesion of Unstable Angina

patients had no history of instability within two months (group B). TypeT lesions, defined as eccentricnarrowing I ntracoronary occlusive thrombus with or without rupture of an atherosclerotic plaque has been established as the usual cause of myocardial infarction (MI).1.2 Coronary arteriographic studies during acute

Prevalence and Distribution of the Third Coronary Artery in

SUMMARY: A third coronary artery (TCA) has been defined as a direct branch from the right aortic sinus (RAS) that contributes to the vascularization of the infundibulum of the right ventricle (RV). The distribution of this artery may be important in surgical procedures and in understanding the extent and progression of acute myocardial infarction.

Ventriculography and Coronary Arteriography in the Acutely

The two patients in group C had acute congestive heart failure that responded poorly to medical therapy. They were studied to exclude coronary arterial disease. Both these patients did not have a recent myocardial infarction. All patients were evaluated for a history of allergy, pre­

Mutational analysis of Kir6.1 in Japanese patients with

obtained from all patients before the study. This study population included 19 Japanese patients with CSA (10 men and 9 women with a mean age of 61±14 years). The coronary arteriographic study was performed in all patients. Coronary spasm, defined as total or subtotal occlusion or severe vaso-constriction of the coronary artery associated with

Anterior S-T changes during acute inferior myocardial infarction

Acute inferior myocardial infarction was diagnosed on the basis of typical chest pain, S-T segment elevation, evolutionary Q waves (0.04 set or more in duration) in at least two of leads II, III and aVF, and serum enzyme changes.

Surgical treatment of aneurysms of the inferior left

myocardial infarction associated with coronary artery disease. The remaining inferior wall aneurysm was subannular in relation to the mitral valve and resulted from previous infective endocarditis that penetrated the posterior mitral anulus. The classification of aneurysms in this report was determined by pathological findings observed at opera

Experience With the Starr-Edwards Aortic Valve

is of interest that the patients listed in Table 4 include a 30-year-old man who, after discharge from the hospital, died unexpectedly from demonstrable occlusion of the right coronary artery and acute subintimal hemorrhage (these findings were established at postmortem examination). Another patient, a 58-year-old man Table 4.

Unstable Angina Pectoris: Comparison with the National

fered a previous myocardial infarction. Con- gestive heart failure was present in 14% of the patients, and cardiomegaly was demonstrated on chest roentgenograms in 10% (see Table 1). The average interval between admission to the coronary care unit and operation was 3.9 days. Of those patients who gave a previous

p-nitrophenyl-N-acetyl-l3-glucosamine

thrombolvtic therapy performed within a coronary care unit: one year's experience. ScottMedj1986;31:25-9. 2 Timmis AD, Griffin B, Crick JC, Sowton E. Anisoylated plasminogen streptokinase activator complex in acute myocardial infarction: a placebo-controlled arteriographic coronary recanalization study.] AmCoil Cardiol 1987;10:205-10.

Antithrombotic therapy in coronary artery disease.

Coronary Artery Disease Vlttorlo Bertele and Edwin W. Salzman In the last two decades, improved treatment of elec-trical disturbances and cardiac pump failure have reduced mortality after the ultimate, irreversible myo-cardial event of patients suffering from coronary heart disease: myocardial infarction. However, the

Augmented thromboxane A 2 generation and efficacy of its

xane B, in patients with angina pectoris [5,6] and acute myocardial infarction [6,7]. Previous experimental work has shown that exogenous administration of vasodilat- ing prostaglandins [8-111 and cyclooxygenase blockades [12,13] is effective in preserving myocardial integrity in acute myocardial infarction.

Coral reef atherosclerosis of the suprarenal aorta: A unique

and clinical findings (nine patients) No. % Renovascular hypertension 9 100 Lower extremity ischemia 9 100 Diminished or absent femoral pulses 9 100 Aortic bruit 8 89 Hyperlipidemia 4 44 Congestive heart failure 3 33 Coronary artery disease 3 33 Familial cardiovascular disease 3 33

전벽부 급성심근경색증 환자에서 심전도 V1 유도의 ST 분절 상승과 우관동맥 Conal Branch의

Comparison of coronary arteriographic (CAG) findings between group 1 and group 2. Patients with ST ele-vation less than 1.5 mm in V1 (group 1) have significantly more large conal branches than patients with ST elevation greater than 1.5 mm in V1 (group 2). In group 2, large conal branch was more frequently found in immediate CAG subgroup.

Arteriography findings in coronary sclerosis

Alterations in the coronary arteries at different ages and the most significant arteriographic findings in coronary sclerosis are described. The usefulness of arteriography in the study of the condition is emphasized and the possible surgical value of the data obtained is pointed out. ZUSAMMENFASSUNG Aus einer Reihe von 274 of - of

Atrioventricular block in acute inferior wall myocardial

acute phase of an inferior wall myocardial infarction. These patients were submitted to coronary cinearteriography in the fourth week of hospitalization, independentof clinical status or symptoms. Their mean age was 54.9 ± 9.1 years (range 32 to 67) and 44 were male. Only one patient had a history of previous myocardial infarction. Eleven of

Silent Obstruction of the Coronary T0M0YUKI YAMBE, SHIN-ICHI

characteristics of the patients with silent obstruction of the coronary artery without myocardial infarction in a short time, compared with the patients with unstable angina pectoris, who are considered to become an acute myocardial infarction with severe chest symptom (Raffiendeul et al. 1979).

There Is Much to Be Gained by Discarding Preconceived - JST

TP, et al. Coronary arteriographic findings soon after non-Q-wave myocardial infarction. N Engl J Med 1986; 315: 417 423. 3. Gehrie ER, Reynolds HR, Chen AY, Neelon BH, Roe MT, Gibler WB, et al. Characterization and outcomes of women and men with non-ST-segment elevation myocardial infarction and nonobstructive coronary artery disease

Volume Subject Index - AHA/ASA Journals

Combined effects of acute cerebrovascular ischemia and myo-cardial infarction in arteriosclerotic, male Sprague-Dawley rats, 183 Intracranial arterial aneurysms in infancy and childhood, 85 Platelet aggregation and cyclic nucleotide phosphodiesterase activity in arteriosclerotic patients, 540 Arteritis Angiographic findings in giant cell

Original Article Clinical profile of patients presenting with

17. Saleheen D, Fossard P. CAD risk factors and acute myocardial infarction in Pakistan. Acta Cardiol 2004; 59: 417-24. 18. Ahmad I, Shafique Q. Myocardial infarction under age 40: Risk factors and coronary arteriographic findings. Ann King Edward Med Coll 2003; 9: 262-5. 19. Ishaq M, Beg MS, Ansari SA, Hakeem A, Ali S. Coronary artery disease risk

Magnetic resonance angiography of peripheral runoff vessels

Previous myocardial infarction 15 Previous cerebral vascular accident 8 Smoking 28 Previous vascular surgery 29 Coronary artery disease 18 Renal insuf~ciency 15 Rest pain 28 Gangrene/nonhealing ulcer 27 Severe claudication 6 Failing vascular graft 3 runoff vessels. The purpose of this study was to

Nifedipine in the Treatment of Unstable Angina, Coronary

the pathophysiologic derangements underlying myocardial infarction. It is now accepted that temporary obliteration of coronary blood flow due to increased vessel wall tone, even in the absence of fixed coronary arterial obstruction and stenosis, can lead to myocardial infarction. Most

Unstable angina pectoris Clinical, angiographic, and

unstable angina pectoris, together with a descrip- myocardial infarction was established in 12 patients by tion of the clinical, left ventriculographic, and either review ofprevious records or bythe presence of coronary arteriographic findings, forms the basis diagnostic Qwaves onthe electrocardiogram. for this report.

Diagnostic Value Exercise Electrocardiography Thallium

Onehundredsixty patients, 130menand30women, suspected of CADbut without evidence ofa previous myocardial infarction, were studied. They underwent a multistage maximal exercise test 1 to several days before the arteriographic study. These patients were studied because of the presence ofchest pain. Before the exercise test, the patients were

Evidence for transient limitations in coronary blood flow

stable periods. Consequently, we compared myocardial oxygen demand in 12 patients at the onset of spontaneous pain during unstable angina to myocardial oxygen demand during exercise-induced ischemia after resumption of stable angina, 6-12 weeks later. Myocardial oxygen demand was estimated from values for heart rate (HR), systolic blood

5-year outcome of an interventional strategy in non-ST

myocardial infarction with a routine interventional strategy (odds ratio 0 82, 95% CI 0 72 0 93).2 However, there was an early net increase in the risk of death during the index hospital admission (1 60, 1 14 2 25, p=0 007). The defining of long-term outcomes in patients with acute coronary syndrome is of critical importance. Experience

Identification of the Culprit Artery Involved in Inferior

the right coronary (RCA) or left circumflex (LCX) artery as the responsible vessel in inferior wall acute myocardial infarction (AMI) in 73 patients. A standard 12-lead ECG was performed within 6 h of onset of chest pain. Coronary angiography was per-formed between 1 week and 6 weeks after the infarction. RCA and LCX lesions were

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Coronary Arteriographic Findings Soon after Non-Q-Wave Myocardial Infarction. NEJM , 315(7), 417-423. Steve Nissen European Atherosclerosis Society Meeting April May 2004

Electrocardiography, cineangiography and myocardial

persion studies in patients with chest pain* Studies from 182 patients were reviewed and compared. Findings from the anterior surface indicated close relationships between the abnormal dispersion study and ab­ normal electrocardiographic and arteriographic findings.